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A growing body of evidence suggests that reactive oxygen species (ROS) are implicated in OA pathogenesis by triggering oxidative stress 2,8,9

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In vitro, CD36, FATP, CPT1a, and ACOX1 were significantly downregulated in HK-2 cells treated with COM, recovered after PRMT1 knockdown, and further decreased after PRMT1 overexpression (Fig

The optic nerve head in glaucoma: role of astrocytes in tissue remodelig