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While homozygous C- Glud1 / mice show an increase in excitatory transmission under stress-nave conditions [14], transcriptional abnormalities implicating pyramidal neuron dysfunction in heterozygous CNS- Glud1 +/ mice are triggered by stress

32 Malfunction of the eNOS pathway, such as due to aging, inflammation, oxidative stress, and nutrient deficiencies, results in a cycle of endothelial NO deficiency, dysregulated vascular tone, and endothelial damage

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Reports from real patients show that side effects might happen more or less because doses can differ

Arch Pediatr Adolesc Med 161:35661, PMID: 17404132 Rezaei Z, Saadat I, Saadat M (2012) Association between three genetic polymorphisms of glutathione S -transferase Z1 (GSTZ1) and susceptibility to bipolar disorder