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Bulletin of Experimental Biology and Medicine, 120(9), 1044-1047

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Commercial availability would follow several months after approval, with widespread access likely beginning the following year

Mitochondrial decline is not peripheral to the aging process it is mechanistically central to it, driving cellular energy deficits, oxidative stress accumulation, and downstream damage to DNA, proteins, and lipids

This leads to one of two outcomes: programmed cell death (apoptosis) or a state of irreversible growth arrest known as cellular senescence

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