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Conclusions Nrf2 is a master regulator of several protective stress response pathways and it is not surprising that it became a highly competitive target for pharmacological modulation (Cuadrado et al

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86 In fact, due to the overlap of phenotypes and growing understanding of intersecting biology, it remains controversial that the definition and boundary between syndromic and idiopathic ASD

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however, GHR ablation in broader neural populations did not produce additional effects as compared to AgRP GHR KO mice, emphasizing a crucial role of AgRP neurons mediating GH responses

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