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ATMs have two main phenotypes: M1 macrophages (M1) is considered the culprit of AT inflammation, can participate in glycolysis to rapidly produce ATP, and plays a key role in the initiation and maintenance of systemic inflammation and insulin resistance

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[4] By supplying additional NAD+ to the body, NAD+ Injection may help ensure these energy-producing pathways can proceed optimally, particularly in situations of NAD+ depletion

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However, start with one or the other to understand individual effects before adding complexity