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In addition, both riboflavin and PLP are essential determinants of the kynurenine pathway which influence glutamate receptors, hence, glutamate excitotoxicity potential, as shown in Section Riboflavin as a Determinant of the Kynurenine Pathway. Concerning PD, glutamate excitotoxicity contributes to dopaminergic neuronal loss (84)

2), the combined suppression of LDHB and GPX4 expression significantly increased mitochondrial lipid peroxidation compared to either treatment alone both in HT1080 and A549 cells (Fig

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Only MsrB1 has been characterized as a selenoprotein with one selenocysteine residue in its catalytic site