When NNMT is active, fat cells become metabolically lazy. By blocking it, this peptide reactivates fat cell metabolism, increases NAD+ availability in adipose tissue, and essentially flips a switch from fat-storing to fat-burning at the cellular level
Patients with Normal Intestinal Absorption: Where the oral route is not deemed adequate, initial treatment similar to that for patients with pernicious anemia may be indicated depending on the severity of the deficiency
Extended treatment continued to limit body weight gain and white adipose tissue mass, lowered total plasma cholesterol, and showed measurable accumulation in adipose tissue consistent with the compounds lipophilic profile
Another source of deficiency can be malabsorption issues, where the body is unable to properly absorb B12 from the intestines due to various gastrointestinal disorders
Disclaimer: The FDA does not approve compounded medications for safety, quality, or manufacturing
AOD 9604 causes none of these effects