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Surprisingly, inhibition of GSH synthesis by BSO was unable to change the distribution of cellular GSH, showing a cellular glutathione pattern different from the one shown in the DEM-treated cells

Concurrently, activation of the Nrf2GSH axis enhances the cellular antioxidant capacity, enabling tumor cells to maintain redox homeostasis under targeted therapyinduced oxidative stress, thus facilitating metabolic escape [77]

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Documentation was based on information from full-text publications, including preclinical and clinical trials, meta-analyses, and reviews on this topic

When asked about the results that particularly stood out, Dr Al-Aly highlighted the effect on substance use disorders

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