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Similar oxidative reactions have also been shown to occur at physiological pH (7.4) [70]

(2015) reported that the CP-induced tubular damage could be diminished by either knockout or inhibition of RIPK3 or MLKL activity

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63, 64, 65 We thus posited that AA might exhibit greater inhibitory effects under oxidative conditions

who found that intervertebral discs away from the fracture sides did not develop any MRI signal changes after 1 year

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