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This points to the fact that study designs capturing only the post-acute phase may not take into account valuable explanations for pathogenesis of PASC (132)

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simultaneously suppressing upstream NF-B activation signals to reduce NLRP3 inflammasome priming, decreasing IL-1 and IL-18 release ( (C) Nrf2 pathway: Vitamin D 3 down-regulates the expression of Keap1, a cytoplasmic inhibitor of Nrf2, promotes the nuclear translocation of Nrf2, binds to antioxidant response elements (ARE) ( 4.2 NLRP3 inflammasome Vitamin D 3 effectively inhibits NLRP3 inflammasome assembly and activation through dual mechanisms: blocking NLRP3 deubiquitination ( Figure 2B )

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