Therefore, in this study, we have used human hepatoma HepG2 cancer cells, a commonly used cell culture model, which has been treated with buthionine sulfoximine (BSO), a GSH depleting agent and N-acetylcysteine (NAC), a precursor of GSH synthesis, to study the implications of altered glutathione metabolism in ASA-induced oxidative stress, redox homeostasis and mitochondrial dysfunctions
Additionally, certain medications and genetic predispositions can increase susceptibility to developing hyperpigmentation, making it a multifaceted concern requiring targeted treatment approaches
Importantly, it inhibits melanogenesis by suppressing the activity of tyrosinase, and oral administration of GSH in humans reduces melanin production in the skin
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Snail growth significantly decreased when exposed to microplastics alone and in combination with ibuprofen (p = 0.021
