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El-Rayes BF, Shields AF, Vaishampayan U, Heilbrun LK, Zalupski MM, Philip PA

67 MelliosN.WoodsonJ.GarciaR

Oral pathogens, especially P.g , can upregulate hepatic flavin-containing monooxygenase 3 (FMO3) through their derived products and promote the conversion of TMA to TMAO, while simultaneously disrupting the intestinal barrier and disturbing the gut microbiota structure, forming an oralgutliver pathogenic pathway (Wang et al., 2024

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Other studies have successfully used ethyl chloroacetoacetate as a starting material to produce ethyl (R)-4-chloro-3-hydroxybutyrate with high optical purity via asymmetric catalytic hydrogenation, which is then converted into L-carnitine

Emodin also inhibits NLRP3 expression and reduces pro-inflammatory factors like interleukin-1 (IL-1), IL-6 and TNF-