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456 Inadequate oxygen supply in failing hearts shunts some free fatty acids into nonoxidative pathways, producing toxic lipids such as ceramides and diacylglycerol, which further damage mitochondrial function and exacerbate heart failure progression

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However, this result is expected considering the very high activity of glutaredoxins as deglutathionylating enzymes and a decreased GSSR concentration neither excludes mechanism (ii) or (iii) from Fig