revealed that GPX4 overexpression prevents basophilic leukemia cells in mice from death caused by oxidative damage (Imai et al., 1996), thus, theoretically, GPX4 overexpression could make cells resistant to oxidative stress-induced ferroptosis
In particular, luteal-phase stress sensitivity and neurotransmitter fluctuations interact to promote hedonic food-seeking behavior, thereby altering the quantity and composition of nutrients entering the metabolic system [64]
10.1016/j.jri.2023.103833 64 JamesE
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Pharmacol Ther 234:108117 Apostolova N, Gomez-Sucerquia LJ, Moran A, Alvarez A, Blas-Garcia A, Esplugues J (2010) Enhanced oxidative stress and increased mitochondrial mass during efavirenz-induced apoptosis in human hepatic cells
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