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Using shRNA to silence CD36 in LNCaP prostate cancer cells, the researchers found that treatment with SIM1-Me (10 nM) or ARV-110 (50 nM) led to: 5.6-fold and 19.6-fold reductions, respectively, in cellular drug uptake dramatically impaired target protein degradation markedly reduced cytotoxicity (up to 423-fold) Re-expression of CD36 restored activity, and loss of EEA1 also diminished SIM1-Me and ARV-110 cytotoxicity---confirming a dependence on the Rab5/EEA1 endocytic pathway

Her approach is rooted in evidence, transparency, and long-term patient safety not hype

1) The dehydration dilemma unveiling the connection

This is the inflection point

(GBD 2019 Benign Prostatic Hyperplasia Collaborators) (November 2022)

How Peptide Therapy Works Peptides are short chains of amino acids that act as signaling molecules in the body