1 Introduction Sepsis-associated encephalopathy (SAE) represents a severe complication of sepsis, defined by diffuse brain dysfunction without direct central nervous system (CNS) infection ( Oxidative stress is central to SAE pathogenesis, driven by an imbalance between excessive reactive oxygen species (ROS) production and impaired antioxidant defense systems ( The antioxidant defense system, especially GSH metabolism, is linked to neurodegenerative diseases ( Proteomics and metabolomics enable systematic profiling of protein expression and metabolic signatures
However, these studies either have a small sample size, or the mechanism research is not in-depth enough
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4446 AFM imaging and force spectroscopy The study of the biological interaction between p28 with full-length p63, p73, or p63 DBD was conducted by AFS, according to the procedure reported by Bizzarri et al
These processes consequently increase the glucose uptake rates in muscle and adipose tissues
