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At 1 hr post-transfection, the cells were superinfected with either CAV20-wt or CAV20-BSOr at a moi of 0.5 and incubation was continued in the presence of 0.4 mM BSO

As 2 O 3 induces ROS generation by disrupting the mitochondrial ETC, impairing mitochondrial membrane potentials, and depleting GSH in HeLa and Calu-6 cancer cells [190, 191]

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Its tri-agonist receptor activity supports research into receptor pharmacology, endocrine pathway regulation, lipid metabolism, hepatic signaling, and cross-tissue metabolic communication in controlled laboratory and translational research environments [1][5]

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