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Specific correlations exist, such as TCA with severe steatosis, GCA with inflammation, and total BA/GCA/TCA with fibrosis (139, 140)
This is why IV therapy is so powerful
Nrf2 depletion in macrophages leads to increased foam cell formation, increases the M1 inflammatory phenotype with enhanced expression of pro-inflammatory monocyte chemoattractant protein-1 and interleukin-6, and aggravates atherosclerosis (244, 245)
Extracts from Artemisia argyi (AA) contain various flavonoids and volatile oils and exert cardioprotective effects through anti-inflammatory, antioxidant, and anti-apoptotic mechanisms ( Astragaloside IV (AS-IV), the principal active component of Astragalus membranaceus, exhibits antifibrotic, anti-apoptotic, antioxidant, and energy metabolism-enhancing properties in cardiomyocytes (Wang et al., 2024)
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