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Follicular development comes to a standstill, and androgen conversion to estradiol slows down due to inadequate FSH output
The model suggests that this decrease is caused by the sequestration of Keap1 in the complex with Nrf2 due to its interaction with DEM
[5] That said, as with any bioactive compound used off-label, individual responses vary and medical oversight is important
These findings lend support to the hypothesis that high levels of oxidative stress may contribute to the pathophysiology of depressionthough its effects may be anatomically selective
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