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Hyperhomocyst(e)inemia is a risk factor for arterial endothelial dysfunction in humans
By combining distinct peptide mechanisms, researchers can examine how coordinated modulation of pathways such as nitric oxide signaling , cytokine expression , extracellular matrix remodeling , and stem/progenitor cellassociated responses impacts measurable endpoints including healing kinetics , scar architecture , and functional outcomes in tissue physiology
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Robbins, Recent advances in the discovery of senolytics, Mechanisms of Ageing and Development, Volume 200, 2021, 111587