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138 However, excessive or chronic inflammation exhausts immune cells due to the constant exposure of T antigens, causing dysfunction with the maintained expression of inhibitory receptors such as PD1 and CTLA4, which alter transcription

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NMNH is dephosphorylated into NRH to be transported into the cell through equilibrative nucleoside transporters (ENTs) 35 , where it is phosphorylated back into NMNH by adenosine kinase (AK) 36