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Several mechanisms contributing to increased oxidative stress in the diabetic milieu have been proposed, including glucose auto-oxidation, weakened antioxidant defense systems, metabolic abnormalities, and mitochondrial damage [45,46,47]
Slc6a8-Mediated creatine uptake and accumulation reprogram macrophage polarization via regulating cytokine responses
"It could tell us something about how metastases evolve and originate within an individual," he says
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