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Apparently, insulin regulates hepatic GHR biosynthesis and surface translocation in a reciprocal manner, with surface receptor availability as the net result of the divergent effects

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published in Molecular Brain confirmed endogenous MGF expression in neurogenic brain areas with age-dependent decline, and that transgenic MGF overexpression significantly increased BrdU+ proliferative cell counts in the dentate gyrus and subventricular zone of the hippocampus establishing MGF as a neurogenesis-promoting factor in ageing brain biology and directly relevant to PEG-MGF neurogenesis and neuroprotection research applications