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Further, to infer the mechanism of GSH mediated osmotic protection, they followed a comparative proteomic approach which revealed upregulation of several glycolytic enzymes in GSH-supplemented cells during osmotic challenge

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Taken together, preclinical and clinical studies in neuroblastoma suggest the potential for BSO to enhance L-PAM activity against diseases that use myeloablative dosing of L-PAM and previous investigations with one murine plasmacytoma, 17 and a human MM cell line, 8, 10 demonstrated enhanced activity of L-PAM by BSO