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These findings suggest BPC-157 may modulate neuroinflammation and promote neural tissue preservation through mechanisms consistent with its known cytoprotective activity in other tissue types

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AMPK also inhibits lipid biosynthesis by suppressing transcription factor sterol regulatory element-binding protein 1 (SREBP1) and promoting the action of peroxisome proliferator-activated receptor alpha (PPAR), a nuclear receptor that acts to enhance both mitochondrial and peroxisomal FA -oxidation 23

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