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Chronic treatment with PARP inhibitors increased NAD + levels and mitochondrial respiration in 10-week-old mice and triggered UPR mt and increased mitochondrial respiration and lifespan in worms 109,257

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46 Conversely, adequate IGF1 availability exerts neuroprotective effects by enhancing mitochondrial biogenesis, stabilizing synaptic proteins, and attenuating NFBdependent inflammatory signalling

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