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The ability of fecal microbiota transplantation to reduce inflammation, enhance intestinal barrier integrity through the production of SCFA, and rebalance immune function highlights its potential as an emerging treatment modality, particularly for ME/CFS [162]

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We injected the rAAV-CMV-DIO-(EGFP-U6)-shRNA ( Oprm1 )-WPRE-hGH pA (AAV-DIO-shMOR-EGFP) virus or AAV-DIO-EGFP vector control into the bilateral PVN of VgluT2-Cre mice (Fig

These results confirmed that our AAV-hHPN vector design successfully enabled the overexpression of human hepsin in mouse liver

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