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Lutchmansingh et al., PLOS One, 2018 Glutathione as redox biomarker in mitochondrial disease implications for therapy by G.M

Interest broadened sharply after Yang and colleagues (Cell, 2014) used targeted metabolomic profiling across twelve ferroptosis-inducing small molecules and found that glutathione depletion converges on inactivation of the selenoenzyme glutathione peroxidase 4, placing glutathione availability directly upstream of iron-dependent lipid peroxidation

In concordance with the results of the uptake experiments, the image analysis also verified that the cellular internalization of targeted nanocarriers was significantly higher (2.4-times elevation for 3-PLG-dopa-A-GSH

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ROS are good