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The priority lies in optimizing delivery systems to enhance bioavailability and conducting longer-term trials with structural outcomes as primary endpoints

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After that, the evident changes in mitochondrial activity and finally changes in membrane fluidity (particularly, mitochondrial membranes) appear [8]

The positive correlations in human CSF of Xan and several monoamines including 5-HIAA have been noted earlier (Niklasson et al., 1983) and between UA and 5-HIAA ( PURINERGIC SIGNALING, CLINICAL IMPROVEMENT, AND NEUROLOGICAL DEFICITS Associations between purine metabolites and clinical and neurological symptoms were examined before and after 4w antipsychotic treatment (Yao et al., 2012)

The reason: prolonged continuous exposure downregulates IGF-1 receptor density (receptor desensitization)

What non-neuronal mechanisms should be studied to understand epileptic seizures