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Importantly, amylin-responsive neurons in the AP do not express co-receptors for GLP-1 and GLP-15, suggesting different primary targets of these peptides

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Senolytic therapy alleviates A-associated oligodendrocyte progenitor cell senescence and cognitive deficits in an Alzheimers disease model

Nicotinamide riboside restores cognition through an upregulation of proliferator-activated receptor- coactivator 1 regulated -secretase 1 degradation and mitochondrial gene expression in Alzheimers mouse models

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