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Carnitine, alone, is not the problem: it's its conversion to TMAO The mediating effect of having an (unfavorably) TMA-producing microbiome may also explain the heterogeneous results of previous studies on the link of high(er) carnitine intake (3g+/d), all-cause mortality, heart failure, unstable angina, or myocardial reinfarction in the setting of acute myocardial infarction as it was descibed by Shang & Hui Li

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