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As described, ABX-induced intestinal dysbiosis results in the altered microbial BA metabolism, which destroys the homeostasis between BA synthesis in the liver and BA metabolism in the intestine, restricts the farnesoid X receptor (FXR)/fibroblast growth factor 15 (FGF15) signaling axis in enterocytes with the conjugated BAs being an effective inhibitor of FXR, and further undermines the interplay of FGF15 with fibroblast growth factor receptor 4 (FGFR4) in the hepatocytes (114)

Marashly, [email protected] Specialty section: This article was submitted to Neurodegeneration, a section of the journal Frontiers in Neurology Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers

2023;19:e1011057

GIP receptor activation in the hypothalamus reduces food intake through mechanisms that are distinct from and complementary to GLP-1 pathways