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Alterations in the expression of these genes may disrupt neural plasticity and emotional regulation, thereby contributing to the development of depression

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Additionally, if OCSCs are exposed to inflammatory mediators, undergo mesenchymal endothelial transformation, or experience dysregulation of the redox balance in the tumor microenvironment (TME), they may stimulate the continuous proliferation of OCSCs, potentially leading to OC development (63)

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Associations among glutathione S-transferase T1, M1, and P1 polymorphisms and the risk of oral leukoplakia