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Microenvironment and tumor heterogeneity as pharmacological targets in precision oncology
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However, based also on small histopathologic studies that pointed to a marked expansion of the exocrine and endocrine pancreatic compartments with the use of incretin-based therapies, the former being accompanied by increased proliferation and dysplasia and the latter by -cell hyperplasia with the potential for evolution into neuroendocrine tumors [105], concerns remain [106], and monitoring for and reporting of pancreatic adverse effects will continue [104]
In rodent models of acute ischemic stroke induced by middle cerebral artery occlusion (MCAO), semaglutide treatment resulted in a substantial reduction in the volume of the resultant brain infarct by 48%63% and facilitated improved functional recovery ( 4.4 Metabolic dysfunction and other models Semaglutides capacity to ameliorate cognitive deficits induced by a high-fat diet in murine models underscores its potential to directly mitigate the neurological consequences associated with metabolic disorders ( 4.5 Translational limitations and contradictory evidence Despite substantial neuroprotective evidence observed in numerous animal models, it is imperative to recognize significant translational limitations and some contradictory findings
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