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In addition to direct effects on ion channels, neurotransmitters may bind to G-protein coupled receptors, thereby eliciting cell-signaling effects that could result in metabolic changes (e.g., alterations in activity of various enzymes) within a postsynaptic cell (14)

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With this positive data, two phase III studies are currently underway via the TULIP (NCT02547922) program

GSH and GABA+) in the developing brain

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This finding convincingly postulated that selective neuronal death of CA1 neurons during global ischemia was mediated by the release of synaptic vesicle zinc from a subset of excitatory terminals and its subsequent translocation into vulnerable post-synaptic neurons