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The results highlight nitrosative redox chemistry as a potentially physiologically relevant pathway for dsNNTTR formation and support a model in which controlled thiol modification, rather than generalized oxidative stress, may contribute to pathological TTR crosslinking in vivo
The study authors reinforced the idea that NNMT inhibition could potentially treat obesity and related metabolic comorbidities [6]
The treatment powers Phase II liver detoxification pathways, which turn toxins into water-soluble compounds your body removes through urine or bile
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