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Smirnova IG, Gildeeva GN, Kukes VG

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In the context of atherosclerosis, a comprehensive systems-level analysis of EC metabolic reprogramming provides an important avenue for developing targeted therapeutic interventions aimed at mitigating age-related vascular pathologies [3]

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This hypothesis is consistent with the idea that glutathione is a mechanism for cysteine storage [12], but also helps explain the reason for the -glutamyl cycle and the reason for the short half-life of hepatic GSH