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Targeting mitochondrial iron metabolism suppresses tumor growth and metastasis by inducing mitochondrial dysfunction and mitophagy

doi: 10.1038/s41594-024-01481-6 72 ShutoY.ShibasakiT.WadaK.ParharI.KamegaiJ.SugiharaH.et al

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A study showed that in a murine model of acute radiation syndrome, iron deposition in the spleen was not mitigated, and the expression level of SLC7A11 remained unchanged after captopril treatment[178]

However, subtle differences exist based on their secondary receptor targets and clinical trial safety data