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& Risbridger, G

Interestingly, a study reported that a rapid increase in proBDNF following status epilepticus induced by pilocarpine may be in part due to decreasing its cleavage due to a decrease in tissue plasminogen activator and an increase in plasminogen activator inhibitor-1 (PAI-1), an inhibitor of extracellular and intracellular cleavage, and that proBDNF is initially enhanced in response to the acute phase of epileptogenesis (Thomas et al

Published data shows this mechanism is particularly relevant in NSAID-induced gut damage models, where NO dysregulation contributes significantly to tissue destruction

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[DOI] [PubMed] [Google Scholar] [7].Laissue JA, Chappuis BB, Muller C, Reubi JC, Gebbers JO

They are referenced to provide scientific context for the ingredients and mechanisms discussed