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2011), results in a slow process of healing, particularly when addressing complete ruptures, which often require surgery

doi: 10.1007/s12603-018-1086-5 18

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ROS also promotes osteoclast differentiation through multiple pathways, including MAPK, IP3, and NFkB [18], leading to osteoclast maturation via activation of genes like NFATC1 [19]

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