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Research suggests that it may support tissue repair, angiogenesis, and modulation of inflammatory responses, primarily through interactions with nitric oxide signaling, growth factor pathways, and cellular migration involved in tissue healing

Curr Med Chem 12:1161208, PMID: 15892631 Morris G, Maes M (2014) Mitochondrial dysfunctions in myalgic encephalomyelitis/chronic fatigue syndrome explained by activated immuno-inflammatory, oxidative and nitrosative stress pathways

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Understanding how pregnancy protects against ovarian and endometrial cancer development: Fetal antigens may be involved

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It should also be noted that RBCs lack other NADPH-producing enzymes, and any loss of NADPH cannot be compensated by other pathways