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The structure of the text progresses from an analysis of specific diseases and their common pathways to a discussion of rationally designed neuroprotective strategies, aiming to bridge scientific knowledge and clinical applications and, hopefully, contribute to better patient outcomes in the future
In this study, we examine how diminished GPX4 function may be a critical event in triggering oxytosis/ferroptosis to perpetuate or initiate the neurodegenerative diseases and assess the possible therapeutic importance of oxytosis/ferroptosis in neurodegenerative disorders
Wang et al., 2025a
gently swirl to dissolve without shaking vigorously