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RIPK1 subsequently interacts with RIPK3 to form the necrosome, a key signaling complex that activates the executor kinase MLKL [95, 96]

If the system were truly driven by patient need, it would have. So why hasnt anyone taken the next step

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Gypenoside a-loaded mpeg-plga nanoparticles ameliorate high-glucose-induced retinal microvasculopathy by inhibiting ferroptosis