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Some dysplastic nevi have been found to exhibit unique gene expression patterns, mutation or altered expression of p16 and p53, and increased microsatellite instability [37]

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The abovementioned signalling hub appears to be equally important for the control of -cell function, a notion supported by human genetic data of genome-wide significance unveiling an association between SPHKAP gene variants, random blood glucose levels/T2D, and high BMI