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Eight-week-old Bmal1 lox/lox / Pax8-rtTA /LC-1 mice and their Bmal1 lox/lox littermate mice were treated for 2 weeks with DOX at 2 mg/mL in drinking water along with 20mg/mL sucrose to induce the Cre recombinase inactivation of the Bmal1 ( Arntl ) gene

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however, this medication has some drawbacks in terms of side effects and a narrow therapeutic window ( It has been documented that PA administration provided protection through metal chelation and its antioxidant properties ( The present data based on APAP-induced hepatotoxicity in mice identified that PA treatment for 7 days at a dose of 200 mg/kg/day has critical actions in modulating APAP-induced liver toxicity in mice