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Additionally, the subchronic exposure to Non-M-CS induced lung inflammation, as evidenced by the pathohistological findings (Figure 2), increased lung vascular permeability (Figure 3A), pulmonary infiltration of inflammatory cells (Figures 3B,C), increases in BALF and lung levels of MIP-2 (Figures 3D,E), and increases in lung levels of 4-HNE (Figure 3F)

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95% CI, 0.270.41)

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