Using a molecular labeling method called TUNEL that lets researchers identify dead cells, the researchers reported aging gene activation protected young CSC-treated cells as their aged CSC-treated counterparts, which did not express those genes, died
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Furthermore, a sublethal dose of CSE induced mitochondrial elongation (Figure 5A-H) , that has been observed during autophagy as a compensatory mechanism to produce ATP and sustain viability under stressful conditions (44)
