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The oral presentation has not yet been studied for this indication, but encouraging evidence should be expected in the future

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In neurology, GHK-Cu is being investigated for its potential to limit neurotoxic damage and support nerve tissue recovery, including through the modulation of oxidative stress and inflammatory processes

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As an example, platelets from G6PD-deficient individuals exhibit reduced G6PD enzymatic activity (89) and of NADPH needed for biosynthesis of fatty acids and phospholipids (90) ( Figure 1A-3 )