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Once activated, p53 promotes cell survival and repair of genetic damage (through target genes that mediate cell cycle arrest and facilitate DNA repair) in response to moderate cellular stress and damage

In this study, we investigated the effects of dysregulated glutathione homeostasis, a principal feature of oxidative stress, on TNF-induced hepatotoxicity and its mechanistic implications in NAFLD progression

References Honda, Yasushi, et al

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Key Benefits: Helps repair sun damage from the inside-out

View in: Pubmed Extracellular Vesicles Transfer the Receptor Programmed Death-1 in Rheumatoid Arthritis