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Moreover, 8q chromosomal gains, frequently encompassing MYC amplification, have been correlated with resistance to EGFR blockade, supporting the rationale for combined EGFR and pan-KRAS inhibition as a next-generation therapeutic strategy (33, 244) ( Table 5 )

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5b) resulted in decreased expression of CYP27A1, CYP27B1, and VDR

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